Search results for "myoclonic epilepsy"

showing 10 items of 11 documents

Benign and severe early-life seizures: a round in the first year of life

2018

Abstract Background At the onset, differentiation between abnormal non-epileptic movements, and epileptic seizures presenting in early life is difficult as is clinical diagnosis and prognostic evaluation of the various seizure disorders presenting at this age. Seizures starting in the first year of life including the neonatal period might have a favorable course, such as in infants presenting with benign familial neonatal epilepsy, febrile seizures simplex or acute symptomatic seizures. However, in some cases, the onset of seizures at birth or in the first months of life have a dramatic evolution with severe cerebral impairment. Seizure disorders starting in early life include the “epilepti…

Ohtahara syndromePediatricsmedicine.medical_specialtyEarly onset seizuresFirst year of lifeReviewEpileptic encephalopathieEpileptic encephalopathies03 medical and health sciencesEpilepsyInfantile epilepsy0302 clinical medicineDravet syndromeSeizures030225 pediatricsmedicineHumansEarly myoclonic encephalopathybusiness.industryAge FactorsInfant Newbornlcsh:RJ1-570InfantWest SyndromeSymptomatic seizureslcsh:Pediatricsmedicine.diseaseMyoclonic epilepsyEarly onset seizurebusiness030217 neurology & neurosurgeryItalian Journal of Pediatrics
researchProduct

Lack of SCN1A Mutations in Familial Febrile Seizures

2002

Summary:  Purpose: Mutations in the voltage-gated sodium channel subunit gene SCN1A have been associated with febrile seizures (FSs) in autosomal dominant generalized epilepsy with febrile seizures plus (GEFS+) families and severe myoclonic epilepsy of infancy. The present study assessed the role of SCN1A in familial typical FSs. Methods: FS families were selected throughout a collaborative study of the Italian League Against Epilepsy. For each index case, the entire coding region of SCN1A was screened by denaturant high-performance liquid chromatography. DNA fragments showing variant chromatograms were subsequently sequenced. Results: Thirty-two FS families accounting for 91 affected indiv…

GAMMA-2-SUBUNITMaleFebrile convulsionsDNA Mutational Analysismedicine.disease_causePolymerase Chain ReactionSodium ChannelsFebrileEpilepsyExonPLUSDNA Mutational AnalysisGene duplicationChildIndex caseChromatography High Pressure LiquidGeneticsChromatographyMutationIdiopathic epilepsyExonsNeurologyIon channelsHigh Pressure LiquidFemaleGeneralized epilepsy with febrile seizures plusMutationsAdultAdolescentGENERALIZED EPILEPSYNerve Tissue ProteinsSeizures FebrileSeizuresGeneticsmedicineHumansFamilybusiness.industryCONVULSIONSGene AmplificationSODIUM-CHANNELmedicine.diseaseGENEDYSFUNCTIONNAV1.1 Voltage-Gated Sodium ChannelFebrile convulsions; Genetics; Idiopathic epilepsy; Ion channels; Mutations; Adolescent; Adult; Child; Chromatography High Pressure Liquid; DNA Mutational Analysis; Exons; Female; Gene Amplification; Humans; Male; Mutation; NAV1.1 Voltage-Gated Sodium Channel; Nerve Tissue Proteins; Polymerase Chain Reaction; Seizures Febrile; Sodium Channels; FamilyMutationMyoclonic epilepsyNeurology (clinical)businessEpilepsia
researchProduct

Panniculitis due to potassium bromide.

1998

Potassium bromide again is well known to be surprisingly effective in patients with severe myoclonic epilepsy in infants (SME). Rare side effects on the skin reappeared, such as the febrile nodular panniculitis (Weber-Christian syndrome). In 1993 we described the first three cases of necrotizing panniculitis and introduced the term 'halogen panniculitis'. It is a systemic disease with crops of subcutaneous nodules, fever, elevated sedimentation rate, hepatosplenomegalia, and abdominal pain. Later severe necrosis of the skin and adipose tissue may happen with deep ulcerations. History and course of five cases, described in this paper, suggest either an allergy or toxic reason. Histologic pic…

BromidesMaleSystemic diseaseAbdominal painPathologymedicine.medical_specialtyAllergyNecrosisPanniculitisAdolescentPotassium CompoundsAdipose tissueInflammationSkin DiseasesDevelopmental NeuroscienceRecurrenceSkin UlcermedicineHumansChildRetrospective StudiesEpilepsybusiness.industryGeneral Medicinemedicine.diseaseChild PreschoolPediatrics Perinatology and Child HealthMyoclonic epilepsyAnticonvulsantsFemaleNeurology (clinical)medicine.symptomPanniculitisbusinessBraindevelopment
researchProduct

Benign myoclonic epilepsy in infancy: neuropsychological and behavioural outcome

2003

Benign myoclonic epilepsy in infancy (BMEI) is a rare syndrome of idiopathic generalized epilepsies with onset below 3 years of age. It has been reported that BMEI is associated with a good prognosis, however, recently some studies suggest less favourable neuropsychological outcome. We report a long-term follow-up of seven patients with BMEI. Seizure outcome and neuropsychological, cognitive, and behavioural evolution were discussed for each of them. At the end of follow-up, 86% of children showed neuropsychological and intellectual disorders: two children had mental retardation, three patients achieved a borderline IQ and one normal but low IQ. All but one displayed neuropsychological disa…

MalePediatricsmedicine.medical_specialtyDevelopmental DisabilitiesEpilepsies MyoclonicNeuropsychological TestsBorderline intellectual functioningCognitionDevelopmental NeurosciencemedicineRare syndromeHumansAge of OnsetPsychiatryChildNeuropsychological outcomeBenign myoclonic epilepsy in infancyNeuropsychologyLanguage impairmentBehavioural outcomeCognitionElectroencephalographyGeneral Medicinemedicine.diseaseFine motor skillOnset ageChild PreschoolPediatrics Perinatology and Child HealthMyoclonic epilepsyFemaleNeurology (clinical)Good prognosisPsychology
researchProduct

West syndrome followed by juvenile myoclonic epilepsy: a coincidental occurrence?

2012

West syndrome followed by juvenile myoclonic epilepsy: a coincidental occurrence? is an age-dependent epilepsy with onset peak in the first year of life. According to the ILAE classification, the etiology of WS could be symptomatic or cryptogenic. An idiopathic etiology was considered too. In literature, there was never previously described a transition from WS to JME. Methods: The proband, (male) was referred to our Department at the age of 8 months because he showed clusters of symmetric spasms. Interictal EEG recording displayed an hypsarrhythmic pattern. The clinical and EEG data suggested WS diagnosis. At 1 year of age increasing long and thick hair in both elbow regions was observed. …

Settore M-PSI/08 - Psicologia ClinicaWest syndrome Juvenile myoclonic epilepsySettore MED/39 - Neuropsichiatria Infantile
researchProduct

West syndrome followed by juvenile myoclonic epilepsy: a coincidental occurrence?

2013

Background: West syndrome is an age-dependent epilepsy with onset peak in the first year of life whose aetiology may be symptomatic or cryptogenic. Long-term cognitive and neurological prognosis is usually poor and seizure outcome is also variable. Over the past two decades a few patients with favourable cognitive outcome and with total recovery from seizures were identified among the cryptogenic group suggesting an idiopathic aetiology. Recent research has described two children with idiopathic WS who later developed a childhood absence epilepsy. Case presentation: We reviewed the medical records of patients with West syndrome admitted to the our Child Neuropsychiatry Unit in the last 15 y…

Malemedicine.medical_specialtyPediatricsNeurologyLevetiracetamAdolescentHairy elbows syndromeMyoclonic JerkClinical NeurologyCase ReportEpilepsyChildhood absence epilepsyJuvenile myoclonic epilepsySettore M-PSI/08 - Psicologia ClinicamedicineHumansEpilepsy evolutionPsychiatrySettore M-PSI/02 - Psicobiologia E Psicologia Fisiologicabusiness.industryGenetic predispositionMyoclonic Epilepsy JuvenileBrainInfantWest SyndromeGeneral MedicineWest syndromemedicine.diseaseMagnetic Resonance ImagingPiracetamSettore MED/39 - Neuropsichiatria InfantileWest syndrome Juvenile myoclonic epilepsy Epilepsy evolution Genetic predisposition Hairy elbows syndromeDisease ProgressionMyoclonic epilepsyNeurology (clinical)LevetiracetamJuvenile myoclonic epilepsybusinessSpasms Infantilemedicine.drugBMC neurology
researchProduct

Delineating a new critical region for juvenile myoclonic epilepsy at the 22q11.2 chromosome.

2013

No abstract available

GeneticsChromosomes Human Pair 21Myoclonic Epilepsy JuvenileChromosome Disordersmyoclonic epilepsy 22q11.2 chromosomeBiologymedicine.diseaseBehavioral NeuroscienceEpilepsySettore MED/38 - Pediatria Generale E SpecialisticaNeurologyChromosome (genetic algorithm)rab GTP-Binding ProteinsMutationmedicineHumansNeurology (clinical)Juvenile myoclonic epilepsyEpilepsybehavior : EB
researchProduct

The first case of myoclonic epilepsy in a child with a de novo 22q11.2 microduplication

2011

Chromosome 22, particularly the q11.2 sub-band, has long been recognized as responsible for multiple congenital anomaly disorders. In particular, its susceptibility to subtle microdeletions or, more rarely, microduplications has been attributed to the presence of several low-copy repeats spanning the region as mediators of nonallelic homologous recombination that result in 22q11.2 rearrangements. While recent data suggest that the frequency of 22q11.2 microduplications could be approximately half of all deletions, now only 50 unrelated cases have been reported thus far. However, it is reasonable to suppose that microduplications of 22q11.2 may be largely undetected as a result of a less-dis…

MaleChromosomes Human Pair 22Non-allelic homologous recombinationEpilepsies MyoclonicMultiple congenital anomalyBiologyRAB36 genemyoclonic epilepsySettore MED/38 - Pediatria Generale E SpecialisticaChromosome DuplicationGene duplicationClinical heterogeneityGeneticsmedicineHumansChildIn Situ Hybridization FluorescenceGenetics (clinical)GeneticsComparative Genomic HybridizationFaciesmedicine.diseaseMild learning difficultiesdevelopmental delayPhenotypeSettore MED/03 - Genetica MedicaChild PreschoolMyoclonic epilepsynonallelic homologous recombinationChromosome 2222q11.2 microduplicationComparative genomic hybridizationAmerican Journal of Medical Genetics Part A
researchProduct

Giant subcutaneous lipomatosis in Myoclonic Epilepsy with Ragged Red Fibers syndrome: The first literature report of “laparoscopic” excision

2020

Background MERRF (Myoclonic Epilepsy with Ragged Red Fibers) is a rare multisystem clinical disorder due to a pathogenic variant of the mitochondrial DNA. Rare symptoms of MERRF include multiple systemic lipomatosis with symmetric uncapsulated lipomas occurs generally on the posterior cervical region and upper back. Methods We report the clinical case of a 46 years-old Caucasian man with MERRF syndrome who presented with MSL on the right cervical region. We decided to perform a mini-invasive approach using laparoscopic instruments. We performed a 10-mm incision about 5 cm under the mass and conducted a blunt dissection of the subcutaneous plane. Then we positioned a 10-mm port and started C…

Laparoscopic surgerymedicine.medical_specialtySubcutaneous lipomatosisbusiness.industryMERRF syndromemedicine.medical_treatmentLipomatosisMini-invasive surgeryMERRF syndromelcsh:Surgerylcsh:RD1-811Laparoscopic excisionLaparoscopic surgerymedicine.diseaseSurgeryMini invasive surgerySettore MED/18 - Chirurgia GeneraleRagged-red fibersSubcutaneous lipomatosismedicineMyoclonic epilepsySurgeryVideoassisted surgerybusiness
researchProduct

Alteration of dopamine D2/D3 receptor binding in patients with juvenile myoclonic epilepsy

2010

Summary Purpose:  To quantify extrastriatal and striatal D2/D3 receptor binding in patients with juvenile myoclonic epilepsy (JME) using the high-affinity dopamine D2/D3 receptor positron emission tomography (PET) ligand 18F-Fallypride ([18F]FP). Methods:  Twelve patients with JME and 21 age-matched control subjects were studied. Dynamic images (180 min) were acquired after injection of [18F]FP. Patients had been seizure-free of all seizure types for at least 10 days before scanning. Parametric images of binding potential (BP) were created using the simplified reference tissue model. The images were stereotactically normalized using a ligand-specific template. We performed a voxel-based ana…

medicine.medical_specialtybusiness.industryPutamenVentral striatumCaudate nucleusmedicine.diseaseTemporal lobemedicine.anatomical_structureEndocrinologynervous systemNeurologyDopamine receptor D3Dopamine receptor D2Internal medicineBasal gangliamedicineNeurology (clinical)Juvenile myoclonic epilepsybusinessEpilepsia
researchProduct